Selective compartmental dominance: an explanation for a
noninfectious, multifactorial etiology for acquired immune
deficiency syndrome (AIDS), and a rationale for ozone therapy and
other immune modulating therapies.
Shallenberger F
Med Hypotheses, 1998 Jan, 50:1, 67-80
Abstract
The most widely accepted etiological explanation for acquired
immune deficiency syndrome (AIDS) currently invokes an infectious
model involving the human immunodeficiency virus (HIV). Because
this infectious model has failed to meet any conventional criteria
for establishing microbial causation, this theory still relies on
the high, though not perfect, statistical correlation linking
presence of HIV antibodies with patients diagnosed with, and at
risk for the syndrome. Many scientists and clinicians now doubt
the HIV theory, though, and propose instead a multifactorial
causation similar to that seen in cancer and heart disease. In
order to discard the HIV model, however, it is necessary to
explain the high statistical correlation mentioned above. Recent
studies involving cellular mediated immunity and cytokine
modulation may explain this statistical relationship without the
need to invoke infectious causation, by suggesting certain
functional characteristics and feedback loops in the immune system
which the author calls selective compartmental dominance (SCD).
SCD provides a model in which chronic dominance of the humoral
immune compartment secondary to chronic high-dose antigenic
challenge results in chronic suppression of the cellular immune
compartment. This model predicts that even HIV-negative members of
the risk groups are susceptible to AIDS, assigns no special causal
role for HIV in AIDS, and suggests a rational course of nontoxic
therapy that can potentially reverse cases in the earlier stages.
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